Lead ?non-biodegradable, soft, malleable, as well as heat and corrosion resistant ?is environmentally omnipresent. Its known properties make it an ideal metal for automobiles, paint, smelting, ceramics, and plastics. Not many years ago, it was also utilized in the toy industry. Unfortunately, lead is toxic to humans. Humans neither need lead nor derive benefits from it. Although lead toxicity has been a global concern since the industrial revolution in the late 1800s, civilization has been unable to prevent or control it satisfactorily. Overall incidence of lead poisoning among American children has fallen from 4.4% in the early 1990s to 1.4% in 2004. In 2002, around 10 out of every 100,000 of adults had lead toxicity. Venous blood lead levels (BLLs) of 10 mcg/dL and 25 mcg/dL were considered toxic in children and adults, respectively. But, since any level of lead can cause toxicity, the CDC announced a new, lower reference value for children in June 2012: 5 mcg/dL. Infants and children absorb a higher fraction of lead than adults do when exposed, increasing their vulnerability. Approximately 450,000 American children have BLLs >5 mcg/dL. Consequently, lead poisoning is still a problem. Lead exposure can start with prenatal maternal-fetal transmission. Outside the womb, children may inhale (or eat) lead dust, often present in street debris, soil, and most frequently, aged house paint. Lead-based paint was phased out in the 1970s, lowering, but not eliminating, risk of exposure. Old pipes sometimes leach lead into drinking water. Lead hazards are disproportionately found in low-income housing. Adults rarely develop lead poisoning, but risk is increased for industrial workers who use or manufacture lead-based products. Health care providers use many tests to identify lead poisoning. In addition to the BLL, a blood smear may show basophilic stippling ribosomal clusters. Increased urinary aminolaevulinic acid concentrations are also reliable indicators. Plain film radiographs can reveal visible lead lines in patients' long bones. Astute clinicians sometimes diagnose lead poisoning after seeing a blue line along patients' gums (Burton's line) that forms when lead reacts with sulfur ions released by oral bacteria. Lead affects every organ system and causes an unpredictable variety of symptoms. The nervous system is most sensitive (centrally in children, peripherally in adults), but lead affects hematopoietic, hepatic, and renal systems, producing serious disorders. Acute lead poisoning's classic symptoms include colic, encephalopathy, anemia, neuropathy, and Fanconi syndrome (abnormal glucose, phosphates, and amino acid excretion). Sometimes, classic signs and symptoms are absent, confusing the clinical picture.
Which of the following best explains the "many tests [used] to identify lead poisoning?"
A. the disproportionate number of lead hazards found in low-income housing
B. the 450,000 American children who have BLLs > 5 mcg/dL
C. the unpredictable variety, and occasional absence, of symptoms
D. lead's environmental omnipresence
Lead ?non-biodegradable, soft, malleable, as well as heat and corrosion resistant ?is environmentally omnipresent. Its known properties make it an ideal metal for automobiles, paint, smelting, ceramics, and plastics. Not many years ago, it was also utilized in the toy industry. Unfortunately, lead is toxic to humans. Humans neither need lead nor derive benefits from it. Although lead toxicity has been a global concern since the industrial revolution in the late 1800s, civilization has been unable to prevent or control it satisfactorily. Overall incidence of lead poisoning among American children has fallen from 4.4% in the early 1990s to 1.4% in 2004. In 2002, around 10 out of every 100,000 of adults had lead toxicity. Venous blood lead levels (BLLs) of 10 mcg/dL and 25 mcg/dL were considered toxic in children and adults, respectively. But, since any level of lead can cause toxicity, the CDC announced a new, lower reference value for children in June 2012: 5 mcg/dL. Infants and children absorb a higher fraction of lead than adults do when exposed, increasing their vulnerability. Approximately 450,000 American children have BLLs >5 mcg/dL. Consequently, lead poisoning is still a problem. Lead exposure can start with prenatal maternal-fetal transmission. Outside the womb, children may inhale (or eat) lead dust, often present in street debris, soil, and most frequently, aged house paint. Lead-based paint was phased out in the 1970s, lowering, but not eliminating, risk of exposure. Old pipes sometimes leach lead into drinking water. Lead hazards are disproportionately found in low-income housing. Adults rarely develop lead poisoning, but risk is increased for industrial workers who use or manufacture lead-based products. Health care providers use many tests to identify lead poisoning. In addition to the BLL, a blood smear may show basophilic stippling ribosomal clusters. Increased urinary aminolaevulinic acid concentrations are also reliable indicators. Plain film radiographs can reveal visible lead lines in patients' long bones. Astute clinicians sometimes diagnose lead poisoning after seeing a blue line along patients' gums (Burton's line) that forms when lead reacts with sulfur ions released by oral bacteria. Lead affects every organ system and causes an unpredictable variety of symptoms. The nervous system is most sensitive (centrally in children, peripherally in adults), but lead affects hematopoietic, hepatic, and renal systems, producing serious disorders. Acute lead poisoning's classic symptoms include colic, encephalopathy, anemia, neuropathy, and Fanconi syndrome (abnormal glucose, phosphates, and amino acid excretion). Sometimes, classic signs and symptoms are absent, confusing the clinical picture.
Why are infants and children more vulnerable to lead poisoning?
A. They can absorb a greater fraction of lead than adults with the same exposure.
B. The CDC announced a new, lower reference value for children.
C. They can become exposed through prenatal maternal-fetal transmission.
D. Because 450,000 American children have BLLs >5 mcg/dL.
Lead ?non-biodegradable, soft, malleable, as well as heat and corrosion resistant ?is environmentally omnipresent. Its known properties make it an ideal metal for automobiles, paint, smelting, ceramics, and plastics. Not many years ago, it was also utilized in the toy industry. Unfortunately, lead is toxic to humans. Humans neither need lead nor derive benefits from it. Although lead toxicity has been a global concern since the industrial revolution in the late 1800s, civilization has been unable to prevent or control it satisfactorily. Overall incidence of lead poisoning among American children has fallen from 4.4% in the early 1990s to 1.4% in 2004. In 2002, around 10 out of every 100,000 of adults had lead toxicity. Venous blood lead levels (BLLs) of 10 mcg/dL and 25 mcg/dL were considered toxic in children and adults, respectively. But, since any level of lead can cause toxicity, the CDC announced a new, lower reference value for children in June 2012: 5 mcg/dL. Infants and children absorb a higher fraction of lead than adults do when exposed, increasing their vulnerability. Approximately 450,000 American children have BLLs >5 mcg/dL. Consequently, lead poisoning is still a problem. Lead exposure can start with prenatal maternal-fetal transmission. Outside the womb, children may inhale (or eat) lead dust, often present in street debris, soil, and most frequently, aged house paint. Lead-based paint was phased out in the 1970s, lowering, but not eliminating, risk of exposure. Old pipes sometimes leach lead into drinking water. Lead hazards are disproportionately found in low-income housing. Adults rarely develop lead poisoning, but risk is increased for industrial workers who use or manufacture lead-based products. Health care providers use many tests to identify lead poisoning. In addition to the BLL, a blood smear may show basophilic stippling ribosomal clusters. Increased urinary aminolaevulinic acid concentrations are also reliable indicators. Plain film radiographs can reveal visible lead lines in patients' long bones. Astute clinicians sometimes diagnose lead poisoning after seeing a blue line along patients' gums (Burton's line) that forms when lead reacts with sulfur ions released by oral bacteria. Lead affects every organ system and causes an unpredictable variety of symptoms. The nervous system is most sensitive (centrally in children, peripherally in adults), but lead affects hematopoietic, hepatic, and renal systems, producing serious disorders. Acute lead poisoning's classic symptoms include colic, encephalopathy, anemia, neuropathy, and Fanconi syndrome (abnormal glucose, phosphates, and amino acid excretion). Sometimes, classic signs and symptoms are absent, confusing the clinical picture. The inclusion of what evidence would best support the author's claim in the first paragraph that "civilization has been unable to prevent or control it satisfactorily."
A. A quote from a well-respected doctor.
B. A survey of 10,000 randomly selected people.
C. A collection of statistics stating the number of deaths from lead poisoning.
D. A series of anecdotes describing lead poisoning.
Lead ?non-biodegradable, soft, malleable, as well as heat and corrosion resistant ?is environmentally omnipresent. Its known properties make it an ideal metal for automobiles, paint, smelting, ceramics, and plastics. Not many years ago, it was also utilized in the toy industry. Unfortunately, lead is toxic to humans. Humans neither need lead nor derive benefits from it. Although lead toxicity has been a global concern since the industrial revolution in the late 1800s, civilization has been unable to prevent or control it satisfactorily. Overall incidence of lead poisoning among American children has fallen from 4.4% in the early 1990s to 1.4% in 2004. In 2002, around 10 out of every 100,000 of adults had lead toxicity. Venous blood lead levels (BLLs) of 10 mcg/dL and 25 mcg/dL were considered toxic in children and adults, respectively. But, since any level of lead can cause toxicity, the CDC announced a new, lower reference value for children in June 2012: 5 mcg/dL. Infants and children absorb a higher fraction of lead than adults do when exposed, increasing their vulnerability. Approximately 450,000 American children have BLLs >5 mcg/dL. Consequently, lead poisoning is still a problem. Lead exposure can start with prenatal maternal-fetal transmission. Outside the womb, children may inhale (or eat) lead dust, often present in street debris, soil, and most frequently, aged house paint. Lead-based paint was phased out in the 1970s, lowering, but not eliminating, risk of exposure. Old pipes sometimes leach lead into drinking water. Lead hazards are disproportionately found in low-income housing. Adults rarely develop lead poisoning, but risk is increased for industrial workers who use or manufacture lead-based products. Health care providers use many tests to identify lead poisoning. In addition to the BLL, a blood smear may show basophilic stippling ribosomal clusters. Increased urinary aminolaevulinic acid concentrations are also reliable indicators. Plain film radiographs can reveal visible lead lines in patients' long bones. Astute clinicians sometimes diagnose lead poisoning after seeing a blue line along patients' gums (Burton's line) that forms when lead reacts with sulfur ions released by oral bacteria. Lead affects every organ system and causes an unpredictable variety of symptoms. The nervous system is most sensitive (centrally in children, peripherally in adults), but lead affects hematopoietic, hepatic, and renal systems, producing serious disorders. Acute lead poisoning's classic symptoms include colic, encephalopathy, anemia, neuropathy, and Fanconi syndrome (abnormal glucose, phosphates, and amino acid excretion). Sometimes, classic signs and symptoms are absent, confusing the clinical picture.
The passage as a whole primarily serves to:
A. outline the prevalence of lead and provide a description of lead poisoning, its causes, diagnosis, and effects.
B. raise awareness of the dangers of lead poisoning.
C. describe the causes and effects of lead poisoning.
D. detail the history of lead and lead poisoning.
Lead ?non-biodegradable, soft, malleable, as well as heat and corrosion resistant ?is environmentally omnipresent. Its known properties make it an ideal metal for automobiles, paint, smelting, ceramics, and plastics. Not many years ago, it was also utilized in the toy industry. Unfortunately, lead is toxic to humans. Humans neither need lead nor derive benefits from it. Although lead toxicity has been a global concern since the industrial revolution in the late 1800s, civilization has been unable to prevent or control it satisfactorily. Overall incidence of lead poisoning among American children has fallen from 4.4% in the early 1990s to 1.4% in 2004. In 2002, around 10 out of every 100,000 of adults had lead toxicity. Venous blood lead levels (BLLs) of 10 mcg/dL and 25 mcg/dL were considered toxic in children and adults, respectively. But, since any level of lead can cause toxicity, the CDC announced a new, lower reference value for children in June 2012: 5 mcg/dL. Infants and children absorb a higher fraction of lead than adults do when exposed, increasing their vulnerability. Approximately 450,000 American children have BLLs >5 mcg/dL. Consequently, lead poisoning is still a problem. Lead exposure can start with prenatal maternal-fetal transmission. Outside the womb, children may inhale (or eat) lead dust, often present in street debris, soil, and most frequently, aged house paint. Lead-based paint was phased out in the 1970s, lowering, but not eliminating, risk of exposure. Old pipes sometimes leach lead into drinking water. Lead hazards are disproportionately found in low-income housing. Adults rarely develop lead poisoning, but risk is increased for industrial workers who use or manufacture lead-based products. Health care providers use many tests to identify lead poisoning. In addition to the BLL, a blood smear may show basophilic stippling ribosomal clusters. Increased urinary aminolaevulinic acid concentrations are also reliable indicators. Plain film radiographs can reveal visible lead lines in patients' long bones. Astute clinicians sometimes diagnose lead poisoning after seeing a blue line along patients' gums (Burton's line) that forms when lead reacts with sulfur ions released by oral bacteria. Lead affects every organ system and causes an unpredictable variety of symptoms. The nervous system is most sensitive (centrally in children, peripherally in adults), but lead affects hematopoietic, hepatic, and renal systems, producing serious disorders. Acute lead poisoning's classic symptoms include colic, encephalopathy, anemia, neuropathy, and Fanconi syndrome (abnormal glucose, phosphates, and amino acid excretion). Sometimes, classic signs and symptoms are absent, confusing the clinical picture.
In the first sentence, "omnipresent" most nearly means?
A. extensive
B. unlimited
C. infinite
D. pervasive
Coughs that linger after a cold or sinus problem cause constant disruption in the home, school, and workplace. Often, these dry, nonproductive coughs become increasingly troublesome although other symptoms ?fever, congestion, and fatigue ?resolved days or weeks ago. This stubborn cough persists for weeks, and plagues its victim and the victim's family night and day. The diagnosis might be a common, but overlooked cause of lingering cough: atypical pneumonia caused by mycoplasma. Mycoplasma ? pleomorphic bacteria that lack a cell wall ?are the smallest and simplest self-replicating organisms known to humans. They probably evolved from gram-positive, walled eubacteria by degenerative evolution. Smaller than amoebas, these 0.1-micrometer organisms grow and reproduce slowly and require no oxygen or host cell. They also change shapes asymmetrically, appearing as long, thin filaments, tiny spheres, or branches. Scientists have identified more than 100 mycoplasma species. Fifteen species are known to live in humans, most as normal symbiotic flora. Mycoplasma pneumoniae, previously called "walking pneumonia," is pathogenic in humans. Mycoplasma pneumoniae glides freely and uses its specialized filamentous tips to burrow between cilia within the respiratory epithelium, causing the respiratory epithelial cells to slough. It also produces hydrogen peroxide, which causes initial cell disruption in the respiratory tract and damages erythrocyte membranes. Researchers have determined that more than 40% of infants younger than 1 year old have had a mycoplasma infection. By age 5, approximately 65% of children have been infected. Nearly all adults have been infected at least once, often repeatedly. Mycoplasma pneumonia usually affects people younger than 40 years of age. The highest incidence is found in the 5- to 9-year age group. The risk of contracting mycoplasma pneumonia is greatest for people who live or work in crowded areas, such as daycare facilities, schools, homeless shelters, long-term care units, and military and prison environments. However, many people who develop mycoplasma infections have no identifiable risk factor. Most mycoplasma infections cause mild to moderate clinical symptoms, but the infection incubates over 3 weeks and can last weeks without treatment. This infection cannot be diagnosed based on symptoms alone; laboratory testing is essential. Infection can also cause ear infections, sinus infections, bronchitis, croup, severe sore throats, infectious asthma, and 1 type of the common cold. When mycoplasma infects children, about 25% of them develop nausea, vomiting, or diarrhea.
Which of the following is NOT true about mycoplasma infection?
A. It can be contracted without an identifiable risk factor.
B. It has an incubation period of over three weeks.
C. It can be diagnosed without laboratory testing.
D. It can cause nausea, vomiting, or diarrhea in children.
Coughs that linger after a cold or sinus problem cause constant disruption in the home, school, and workplace. Often, these dry, nonproductive coughs become increasingly troublesome although other symptoms ?fever, congestion, and fatigue ?resolved days or weeks ago. This stubborn cough persists for weeks, and plagues its victim and the victim's family night and day. The diagnosis might be a common, but overlooked cause of lingering cough: atypical pneumonia caused by mycoplasma. Mycoplasma ? pleomorphic bacteria that lack a cell wall ?are the smallest and simplest self-replicating organisms known to humans. They probably evolved from gram-positive, walled eubacteria by degenerative evolution. Smaller than amoebas, these 0.1-micrometer organisms grow and reproduce slowly and require no oxygen or host cell. They also change shapes asymmetrically, appearing as long, thin filaments, tiny spheres, or branches. Scientists have identified more than 100 mycoplasma species. Fifteen species are known to live in humans, most as normal symbiotic flora. Mycoplasma pneumoniae, previously called "walking pneumonia," is pathogenic in humans. Mycoplasma pneumoniae glides freely and uses its specialized filamentous tips to burrow between cilia within the respiratory epithelium, causing the respiratory epithelial cells to slough. It also produces hydrogen peroxide, which causes initial cell disruption in the respiratory tract and damages erythrocyte membranes. Researchers have determined that more than 40% of infants younger than 1 year old have had a mycoplasma infection. By age 5, approximately 65% of children have been infected. Nearly all adults have been infected at least once, often repeatedly. Mycoplasma pneumonia usually affects people younger than 40 years of age. The highest incidence is found in the 5- to 9-year age group. The risk of contracting mycoplasma pneumonia is greatest for people who live or work in crowded areas, such as daycare facilities, schools, homeless shelters, long-term care units, and military and prison environments. However, many people who develop mycoplasma infections have no identifiable risk factor. Most mycoplasma infections cause mild to moderate clinical symptoms, but the infection incubates over 3 weeks and can last weeks without treatment. This infection cannot be diagnosed based on symptoms alone; laboratory testing is essential. Infection can also cause ear infections, sinus infections, bronchitis, croup, severe sore throats, infectious asthma, and 1 type of the common cold. When mycoplasma infects children, about 25% of them develop nausea, vomiting, or diarrhea.
In paragraph 2, "symbiotic" most nearly means
A. independent
B. cooperative
C. divided
D. shared
Coughs that linger after a cold or sinus problem cause constant disruption in the home, school, and workplace. Often, these dry, nonproductive coughs become increasingly troublesome although other symptoms ?fever, congestion, and fatigue ?resolved days or weeks ago. This stubborn cough persists for weeks, and plagues its victim and the victim's family night and day. The diagnosis might be a common, but overlooked cause of lingering cough: atypical pneumonia caused by mycoplasma. Mycoplasma ? pleomorphic bacteria that lack a cell wall ?are the smallest and simplest self-replicating organisms known to humans. They probably evolved from gram-positive, walled eubacteria by degenerative evolution. Smaller than amoebas, these 0.1-micrometer organisms grow and reproduce slowly and require no oxygen or host cell. They also change shapes asymmetrically, appearing as long, thin filaments, tiny spheres, or branches. Scientists have identified more than 100 mycoplasma species. Fifteen species are known to live in humans, most as normal symbiotic flora. Mycoplasma pneumoniae, previously called "walking pneumonia," is pathogenic in humans. Mycoplasma pneumoniae glides freely and uses its specialized filamentous tips to burrow between cilia within the respiratory epithelium, causing the respiratory epithelial cells to slough. It also produces hydrogen peroxide, which causes initial cell disruption in the respiratory tract and damages erythrocyte membranes. Researchers have determined that more than 40% of infants younger than 1 year old have had a mycoplasma infection. By age 5, approximately 65% of children have been infected. Nearly all adults have been infected at least once, often repeatedly. Mycoplasma pneumonia usually affects people younger than 40 years of age. The highest incidence is found in the 5- to 9-year age group. The risk of contracting mycoplasma pneumonia is greatest for people who live or work in crowded areas, such as daycare facilities, schools, homeless shelters, long-term care units, and military and prison environments. However, many people who develop mycoplasma infections have no identifiable risk factor. Most mycoplasma infections cause mild to moderate clinical symptoms, but the infection incubates over 3 weeks and can last weeks without treatment. This infection cannot be diagnosed based on symptoms alone; laboratory testing is essential. Infection can also cause ear infections, sinus infections, bronchitis, croup, severe sore throats, infectious asthma, and 1 type of the common cold. When mycoplasma infects children, about 25% of them develop nausea, vomiting, or diarrhea.
The tone of the passage can best be described as:
A. contemptuous
B. judgmental
C. matter-of-fact
D. solemn
Coughs that linger after a cold or sinus problem cause constant disruption in the home, school, and workplace. Often, these dry, nonproductive coughs become increasingly troublesome although other symptoms ?fever, congestion, and fatigue ?resolved days or weeks ago. This stubborn cough persists for weeks, and plagues its victim and the victim's family night and day. The diagnosis might be a common, but overlooked cause of lingering cough: atypical pneumonia caused by mycoplasma. Mycoplasma ? pleomorphic bacteria that lack a cell wall ?are the smallest and simplest self-replicating organisms known to humans. They probably evolved from gram-positive, walled eubacteria by degenerative evolution. Smaller than amoebas, these 0.1-micrometer organisms grow and reproduce slowly and require no oxygen or host cell. They also change shapes asymmetrically, appearing as long, thin filaments, tiny spheres, or branches. Scientists have identified more than 100 mycoplasma species. Fifteen species are known to live in humans, most as normal symbiotic flora. Mycoplasma pneumoniae, previously called "walking pneumonia," is pathogenic in humans. Mycoplasma pneumoniae glides freely and uses its specialized filamentous tips to burrow between cilia within the respiratory epithelium, causing the respiratory epithelial cells to slough. It also produces hydrogen peroxide, which causes initial cell disruption in the respiratory tract and damages erythrocyte membranes. Researchers have determined that more than 40% of infants younger than 1 year old have had a mycoplasma infection. By age 5, approximately 65% of children have been infected. Nearly all adults have been infected at least once, often repeatedly. Mycoplasma pneumonia usually affects people younger than 40 years of age. The highest incidence is found in the 5- to 9-year age group. The risk of contracting mycoplasma pneumonia is greatest for people who live or work in crowded areas, such as daycare facilities, schools, homeless shelters, long-term care units, and military and prison environments. However, many people who develop mycoplasma infections have no identifiable risk factor. Most mycoplasma infections cause mild to moderate clinical symptoms, but the infection incubates over 3 weeks and can last weeks without treatment. This infection cannot be diagnosed based on symptoms alone; laboratory testing is essential. Infection can also cause ear infections, sinus infections, bronchitis, croup, severe sore throats, infectious asthma, and 1 type of the common cold. When mycoplasma infects children, about 25% of them develop nausea, vomiting, or diarrhea.
Within the context of the passage as a whole, paragraph 2 serves what purpose?
A. Give a general description of mycoplasma.
B. Provide specific details of mycoplasma and how it relates to the common cold.
C. Describe the progression of "walking pneumonia" in humans.
D. Define the characteristics of mycoplasma pneumoniae.
Coughs that linger after a cold or sinus problem cause constant disruption in the home, school, and workplace. Often, these dry, nonproductive coughs become increasingly troublesome although other symptoms ?fever, congestion, and fatigue ?resolved days or weeks ago. This stubborn cough persists for weeks, and plagues its victim and the victim's family night and day. The diagnosis might be a common, but overlooked cause of lingering cough: atypical pneumonia caused by mycoplasma. Mycoplasma ? pleomorphic bacteria that lack a cell wall ?are the smallest and simplest self-replicating organisms known to humans. They probably evolved from gram-positive, walled eubacteria by degenerative evolution. Smaller than amoebas, these 0.1-micrometer organisms grow and reproduce slowly and require no oxygen or host cell. They also change shapes asymmetrically, appearing as long, thin filaments, tiny spheres, or branches. Scientists have identified more than 100 mycoplasma species. Fifteen species are known to live in humans, most as normal symbiotic flora. Mycoplasma pneumoniae, previously called "walking pneumonia," is pathogenic in humans. Mycoplasma pneumoniae glides freely and uses its specialized filamentous tips to burrow between cilia within the respiratory epithelium, causing the respiratory epithelial cells to slough. It also produces hydrogen peroxide, which causes initial cell disruption in the respiratory tract and damages erythrocyte membranes. Researchers have determined that more than 40% of infants younger than 1 year old have had a mycoplasma infection. By age 5, approximately 65% of children have been infected. Nearly all adults have been infected at least once, often repeatedly. Mycoplasma pneumonia usually affects people younger than 40 years of age. The highest incidence is found in the 5- to 9-year age group. The risk of contracting mycoplasma pneumonia is greatest for people who live or work in crowded areas, such as daycare facilities, schools, homeless shelters, long-term care units, and military and prison environments. However, many people who develop mycoplasma infections have no identifiable risk factor. Most mycoplasma infections cause mild to moderate clinical symptoms, but the infection incubates over 3 weeks and can last weeks without treatment. This infection cannot be diagnosed based on symptoms alone; laboratory testing is essential. Infection can also cause ear infections, sinus infections, bronchitis, croup, severe sore throats, infectious asthma, and 1 type of the common cold. When mycoplasma infects children, about 25% of them develop nausea, vomiting, or diarrhea.
Approximately what percentage of known mycoplasma species live in human beings?
A. 2
B. 15
C. 35
D. 100
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